Reply to “comment on ‘onion-skin type of periductular sclerosis in mice with genetic deletion of biliary kindlin-2 as tight junction stabilizer: a pilot experiment indicating a primary sclerosing cholangitis (PSC) phenotype’”

Martina Lukasova; Katharina Weinberger; Ralf Weiskirchen; Wolfgang Stremmel

doi:10.20517/mtod.2024.127

Metabolism and Target Organ Damage ›› 2024, Vol. 4 ›› Issue (4) :48 DOI: 10.20517/mtod.2024.127

Reply to “comment on ‘onion-skin type of periductular sclerosis in mice with genetic deletion of biliary kindlin-2 as tight junction stabilizer: a pilot experiment indicating a primary sclerosing cholangitis (PSC) phenotype’”

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Martina Lukasova, Katharina Weinberger, Ralf Weiskirchen, Wolfgang Stremmel. Reply to “comment on ‘onion-skin type of periductular sclerosis in mice with genetic deletion of biliary kindlin-2 as tight junction stabilizer: a pilot experiment indicating a primary sclerosing cholangitis (PSC) phenotype’”. Metabolism and Target Organ Damage, 2024, 4(4): 48 DOI:10.20517/mtod.2024.127

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References

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[1]	Stremmel W,Weiskirchen R.Phosphatidylcholine passes by paracellular transport to the apical side of the polarized biliary tumor cell line Mz-ChA-1.Int J Mol Sci2019;20:4034 PMCID:PMC6720464

[2]	Stremmel W,Staffer S.Mucosal protection by phosphatidylcholine.Dig Dis2012;30 Suppl 3:85-91

[3]	Stremmel W,Schneider MJ.Genetic mouse models with intestinal-specific tight junction deletion resemble an ulcerative colitis phenotype.J Crohns Colitis2017;11:1247-57 PMCID:PMC5881657

[4]	Fickert P, Pollheimer MJ, Beuers U, et al; International PSC Study Group (IPSCSG). Characterization of animal models for primary sclerosing cholangitis (PSC). J Hepatol 2014;60:1290-303. PMCID:PMC4517670