Dysbiosis as a common pathogenetic mechanism in psoriasis and gastrointestinal diseases

Christos Zavos

Exploration of Musculoskeletal Diseases ›› 2025, Vol. 3 ›› Issue (1) : 100778

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Exploration of Musculoskeletal Diseases ›› 2025, Vol. 3 ›› Issue (1) :100778 DOI: 10.37349/emd.2025.100778
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Dysbiosis as a common pathogenetic mechanism in psoriasis and gastrointestinal diseases
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Psoriasis / gastrointestinal diseases / inflammatory bowel disease / Crohn’s disease / dysbiosis

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Christos Zavos. Dysbiosis as a common pathogenetic mechanism in psoriasis and gastrointestinal diseases. Exploration of Musculoskeletal Diseases, 2025, 3 (1) : 100778 DOI:10.37349/emd.2025.100778

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References

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Zhou L, Zhang M, Wang Y, Dorfman RG, Liu H, Yu T, et al. Faecalibacterium prausnitzii Produces Butyrate to Maintain Th17/Treg Balance and to Ameliorate Colorectal Colitis by Inhibiting Histone Deacetylase 1. Inflamm Bowel Dis. 2018; 24: 1926-40.

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Zhang X, Shi L, Sun T, Guo K, Geng S. Dysbiosis of gut microbiota and its correlation with dysregulation of cytokines in psoriasis patients. BMC Microbiol. 2021; 21: 78.

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Scher JU, Ubeda C, Artacho A, Attur M, Isaac S, Reddy SM, et al. Decreased Bacterial Diversity Characterizes the Altered Gut Microbiota in Patients With Psoriatic Arthritis, Resembling Dysbiosis in Inflammatory Bowel Disease. Arthritis Rheumatol. 2015; 67: 128-39.

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Mao R, Yu Q, Li J. The causal relationship between gut microbiota and inflammatory dermatoses: a Mendelian randomization study. Front Immunol. 2023; 14: 1231848.

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