Causal relationship between endometriosis and inflammatory bowel disease: A Mendelian randomization analyses

Yan Dang , Shutian Zhang

Clinical and Translational Medicine ›› 2024, Vol. 14 ›› Issue (1) : e1496

PDF
Clinical and Translational Medicine ›› 2024, Vol. 14 ›› Issue (1) : e1496 DOI: 10.1002/ctm2.1496
LETTER TO THE JOURNAL

Causal relationship between endometriosis and inflammatory bowel disease: A Mendelian randomization analyses

Author information +
History +
PDF

Cite this article

Download citation ▾
Yan Dang, Shutian Zhang. Causal relationship between endometriosis and inflammatory bowel disease: A Mendelian randomization analyses. Clinical and Translational Medicine, 2024, 14(1): e1496 DOI:10.1002/ctm2.1496

登录浏览全文

4963

注册一个新账户 忘记密码

References

[1]

Taylor HS, Kotlyar AM, Flores VA. Endometriosis is a chronic systemic disease: clinical challenges and novel innovations. The Lancet, 2021;397(10276):839-852.

[2]

Shigesi N, Kvaskoff M, Kirtley S, et al. The association between endometriosis and autoimmune diseases: a systematic review and meta-analysis. Human Reproduction Update, 2019;25(4):486-503.

[3]

Craninx M, D'Haens G, Cokelaere K, et al. Crohn's disease and intestinal endometriosis. European Journal of Gastroenterology & Hepatology, 2000;12(2):217-221.

[4]

Jess T, Frisch M, Jørgensen KT, Pedersen BV, Nielsen NM. Increased risk of inflammatory bowel disease in women with endometriosis: a nationwide Danish cohort study. Gut, 2011;61(9):1279-1283.

[5]

Davies NM, Holmes MV, Davey Smith G. Reading Mendelian randomisation studies: a guide, glossary, and checklist for clinicians. BMJ, k601. 2018.

[6]

Liu JZ, van Sommeren S, Huang H, et al. Association analyses identify 38 susceptibility loci for inflammatory bowel disease and highlight shared genetic risk across populations. Nature Genetics, 2015;47(9):979-986.

[7]

Garitazelaia A, Rueda-Martínez A, Arauzo R, et al. A Systematic Two-Sample Mendelian Randomization Analysis Identifies Shared Genetic Origin of Endometriosis and Associated Phenotypes. Life, 2021;11(1):24.

[8]

Hemani G, Tilling K, Davey Smith G. Orienting the causal relationship between imprecisely measured traits using GWAS summary data. PLOS Genetics, 2017;13(11):e1007081.

[9]

Verbanck M, Chen C-Y, Neale B, Do R. Detection of widespread horizontal pleiotropy in causal relationships inferred from Mendelian randomization between complex traits and diseases. Nature Genetics, 2018;50(5);693-698.

[10]

Hemani G, Zheng J, Elsworth B, et al. The MR-Base platform supports systematic causal inference across the human phenome. ELife, 7. CLOCKSS. 2018.

[11]

Izumi G, Koga K, Takamura M, et al. Involvement of immune cells in the pathogenesis of endometriosis. Journal of Obstetrics and Gynaecology Research, 2018;44(2):191-198. Portico.

[12]

von Lampe B. Differential expression of matrix metalloproteinases and their tissue inhibitors in colon mucosa of patients with inflammatory bowel disease. Gut, 2000;47(1):63-73.

[13]

Salliss ME, Farland LV, Mahnert ND, Herbst-Kralovetz MM. The role of gut and genital microbiota and the estrobolome in endometriosis, infertility and chronic pelvic pain. Human Reproduction Update, 2021;28(1):92-131.

[14]

Felder JB, Korelitz BI, Rajapakse R, Schwarz S, Horatagis AP, Gleim G. Effects of nonsteroidal antiinflammatory drugs on inflammatory bowel disease: a case-control study. The American Journal of Gastroenterology, 2000;95(8):1949-1954.

[15]

Cornish JA, Tan E, Simillis C, Clark SK, Teare J, Tekkis PP. The Risk of Oral Contraceptives in the Etiology of Inflammatory Bowel Disease: A Meta-Analysis. The American Journal of Gastroenterology, 2008;103(9);2394-2400.

RIGHTS & PERMISSIONS

2024 The Authors. Clinical and Translational Medicine published by John Wiley & Sons Australia, Ltd on behalf of Shanghai Institute of Clinical Bioinformatics.

AI Summary AI Mindmap
PDF

158

Accesses

0

Citation

Detail

Sections
Recommended

AI思维导图

/