New data in the study of genetic mechanisms for maintaining "stemness" in human embryonic stem cells

References

[1] Pera M.F., Trounson А.О. Human embryonic stem cells: prospects for development. Development 2004; 131: 5515-25.

[2] Scholer H.R., Balling R., Hatzopoulos A.K. et al., Octamer binding proteins confer transcriptional activity in early mouse embryogenesis. EMBO J. 1989; 8: 2551-7.

[3] Wood H.B., Episkopou V. Comparative expression of the mouse Sox1, Sox2 and Sox3 genes from pre-gastrulation to early somite stages. Meeh. Dev. 1999; 86: 197-201.

[4] Thomson J.A., Itskovitz-Eldor J., Shapiro S.S. et al., Embryonic stem cell lines derived from human blastocysts. Science 1998; 282:1145-7.

[5] Chambers I., Colby D„ Robertson M. et al. Functional expression cloning of Nanog, a pluripotency sustaining factor in embryonic stem cells. Cell 2003; 113: 643-55.

[6] Mitsui K., Tokuzawa Y., Itoh H. et al. The homeoprotein Nanog is required for maintenance of pluripotency in mouse epiblast and ES cells. Cell 2003; 113:631-42.

[7] James D„ Levine A.J., Besser D„ Hemmati-Brivanlou A. TGFbeta/activin/ nodal signaling is necessary for the maintenance of pluripotency in human embryonic stem cells. Development 2005; 132:1273-82.

[8] Dezawa M„ Hoshino M„ Ide C. Treatment of neurodegenerative diseases using adult bone marrow stromal cell-derived neurons. Expert. Opin. Biol. Ther. 2005; 5: 427-35.

[9] Dezawa M„ Ishikawa H., Itokazu Y. etal., Bone marrow stromal cells generate muscle cells and repair muscle degeneration. Science 2005; 309: 314-7.

[10] Gu P„ LeMenuet D„ Chung A.C. et al. Orphan nuclear receptor GCNF is required for the repression of pluripotency genes during retinoic acid-induced embryonic stem cell differentiation. Mol. Cell. Biol. 2005; 25: 8507-19.

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